Studies in mice demonstrated that dietary phosphate (Pi) loading that mimic the level of US adult consumption leads to reduced spontaneous locomotor activity, exercise capacity, and reduced resting metabolic rate when in normal mice by impairing skeletal muscle mitochondrial function and fat oxidation. However, relevance of this findings in humans remains unknown.
Physical Inactivity, Phosphate Overload, Visceral Obesity
Studies in mice demonstrated that dietary phosphate (Pi) loading that mimic the level of US adult consumption leads to reduced spontaneous locomotor activity, exercise capacity, and reduced resting metabolic rate when in normal mice by impairing skeletal muscle mitochondrial function and fat oxidation. However, relevance of this findings in humans remains unknown.
Impact of Dietary Phosphate Excess on Exercise Capacity and Visceral Adiposity
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University of Texas Southwestern, Dallas, Texas, United States, 75209
Researchers look for people who fit a certain description, called eligibility criteria. Some examples of these criteria are a person's general health condition or prior treatments.
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18 Years to 80 Years
ALL
Yes
University of Texas Southwestern Medical Center,
Wanpen Vongpatanasin, MD, PRINCIPAL_INVESTIGATOR, Wanpen.Vongpatanasin@UTSouthwestern.edu
2028-03-30